Article Information
PubMed
Published By
Print ISSN
Online ISSN
History
- Received for publication March 10, 1998
- Accepted for publication September 13, 1998
- Published online March 1, 1999.
Copyright & Usage
© 1999 American Society of Nephrology
Author Information
- MALCOLM A. CUNNINGHAM*,
- XIAO RU HUANG*,
- JOHN P. DOWLING†,
- PETER G. TIPPING* and
- STEPHEN R. HOLDSWORTH*
- * Centre for Inflammatory Diseases, Monash University, Department of Medicine, Monash Medical Centre, Clayton, Victoria, Australia
- †Department of Pathology, Monash University, Alfred Hospital, Prahran, Victoria, Australia.
- Correspondence to Dr. Malcolm Cunningham, Centre for Inflammatory Diseases, Department of Medicine, 5th Floor Block E, Monash Medical Centre, 246 Clayton Road, Clayton, Victoria, 3168, Australia. Phone: 613 9550 5544; Fax: 613 9550 5524; E-mail: malcolmc{at}mmcc1.cc.monash.edu.au
Statistics from Altmetric.com
Article usage
Cited By...
Prominence of Cell-Mediated Immunity Effectors in “Pauci-Immune” Glomerulonephritis
Jump to section
More in this TOC Section
Cited By...
- Tolerogenic Dendritic Cells Attenuate Experimental Autoimmune Antimyeloperoxidase Glomerulonephritis
- Mast Cell Stabilization Ameliorates Autoimmune Anti-Myeloperoxidase Glomerulonephritis
- M2 Macrophage Infiltrates in the Early Stages of ANCA-Associated Pauci-Immune Necrotizing GN
- ANCAs Are Also Antimonocyte Cytoplasmic Autoantibodies
- The immunodominant myeloperoxidase T-cell epitope induces local cell-mediated injury in antimyeloperoxidase glomerulonephritis
- Basic and Translational Concepts of Immune-Mediated Glomerular Diseases
- Th1 and Th17 Cells Induce Proliferative Glomerulonephritis
- Anti-Neutrophil Cytoplasmic Antibodies and Effector CD4+ Cells Play Nonredundant Roles in Anti-Myeloperoxidase Crescentic Glomerulonephritis
- Toll-Like Receptor 2 Agonists Exacerbate Accelerated Nephrotoxic Nephritis
- Anti-Neutrophil Cytoplasm-Associated Glomerulonephritis
- T Cells in Crescentic Glomerulonephritis
- IL-12p40 and IL-18 in Crescentic Glomerulonephritis: IL-12p40 is the Key Th1-Defining Cytokine Chain, Whereas IL-18 Promotes Local Inflammation and Leukocyte Recruitment
- An IL-12-Independent Role for CD40-CD154 in Mediating Effector Responses: Studies in Cell-Mediated Glomerulonephritis and Dermal Delayed-Type Hypersensitivity
- Intrinsic Renal Cell Expression of CD40 Directs Th1 Effectors Inducing Experimental Crescentic Glomerulonephritis
- T Cells and Minimal Change Disease
- The Requirement for Granulocyte-Macrophage Colony-Stimulating Factor and Granulocyte Colony-Stimulating Factor in Leukocyte-Mediated Immune Glomerular Injury
- Human Glomerulonephritis Accompanied by Active Cellular Infiltrates Shows Effector T Cells in Urine
- Glomerulonephritis Induced by Recombinant Collagen IV{alpha}3 Chain Noncollagen Domain 1 Is Not Associated with Glomerular Basement Membrane Antibody: A Potential T Cell-Mediated Mechanism
- Interleukin-12 from Intrinsic Cells Is an Effector of Renal Injury in Crescentic Glomerulonephritis
- Expression of the Chemokine Monocyte Chemoattractant Protein-1 and Its Receptor Chemokine Receptor 2 in Human Crescentic Glomerulonephritis
- Clinical Utility of Testing for Antineutrophil Cytoplasmic Antibodies