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Immunology and Pathology
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Prominence of Cell-Mediated Immunity Effectors in “Pauci-Immune” Glomerulonephritis

MALCOLM A. CUNNINGHAM, XIAO RU HUANG, JOHN P. DOWLING, PETER G. TIPPING and STEPHEN R. HOLDSWORTH
JASN March 1999, 10 (3) 499-506; DOI: https://doi.org/10.1681/ASN.V103499
MALCOLM A. CUNNINGHAM
*
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XIAO RU HUANG
*
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JOHN P. DOWLING
†.
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PETER G. TIPPING
*
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STEPHEN R. HOLDSWORTH
*
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Article Information

vol. 10 no. 3 499-506
DOI 
https://doi.org/10.1681/ASN.V103499
PubMed 
10073600

Published By 
American Society of Nephrology
Print ISSN 
1046-6673
Online ISSN 
1533-3450
History 
  • Received for publication March 10, 1998
  • Accepted for publication September 13, 1998
  • Published online March 1, 1999.

Copyright & Usage 
© 1999 American Society of Nephrology

Author Information

  1. MALCOLM A. CUNNINGHAM*,
  2. XIAO RU HUANG*,
  3. JOHN P. DOWLING†,
  4. PETER G. TIPPING* and
  5. STEPHEN R. HOLDSWORTH*
  1. * Centre for Inflammatory Diseases, Monash University, Department of Medicine, Monash Medical Centre, Clayton, Victoria, Australia
  2. †Department of Pathology, Monash University, Alfred Hospital, Prahran, Victoria, Australia.
  1. Correspondence to Dr. Malcolm Cunningham, Centre for Inflammatory Diseases, Department of Medicine, 5th Floor Block E, Monash Medical Centre, 246 Clayton Road, Clayton, Victoria, 3168, Australia. Phone: 613 9550 5544; Fax: 613 9550 5524; E-mail: malcolmc{at}mmcc1.cc.monash.edu.au

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Journal of the American Society of Nephrology: 10 (3)
Journal of the American Society of Nephrology
Vol. 10, Issue 3
1 Mar 1999
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Prominence of Cell-Mediated Immunity Effectors in “Pauci-Immune” Glomerulonephritis
MALCOLM A. CUNNINGHAM, XIAO RU HUANG, JOHN P. DOWLING, PETER G. TIPPING, STEPHEN R. HOLDSWORTH
JASN Mar 1999, 10 (3) 499-506; DOI: 10.1681/ASN.V103499

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Prominence of Cell-Mediated Immunity Effectors in “Pauci-Immune” Glomerulonephritis
MALCOLM A. CUNNINGHAM, XIAO RU HUANG, JOHN P. DOWLING, PETER G. TIPPING, STEPHEN R. HOLDSWORTH
JASN Mar 1999, 10 (3) 499-506; DOI: 10.1681/ASN.V103499
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More in this TOC Section

  • Contributions of IL-1β and IL-1α to Crescentic Glomerulonephritis in Mice
  • Modulation of Interferon-Induced Genes by Lipoxin Analogue in Anti–Glomerular Basement Membrane Nephritis
  • The Parietal Epithelial Cell: A Key Player in the Pathogenesis of Focal Segmental Glomerulosclerosis in Thy-1.1 Transgenic Mice
Show more Immunology and Pathology

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  • Tolerogenic Dendritic Cells Attenuate Experimental Autoimmune Antimyeloperoxidase Glomerulonephritis
  • Mast Cell Stabilization Ameliorates Autoimmune Anti-Myeloperoxidase Glomerulonephritis
  • M2 Macrophage Infiltrates in the Early Stages of ANCA-Associated Pauci-Immune Necrotizing GN
  • ANCAs Are Also Antimonocyte Cytoplasmic Autoantibodies
  • The immunodominant myeloperoxidase T-cell epitope induces local cell-mediated injury in antimyeloperoxidase glomerulonephritis
  • Basic and Translational Concepts of Immune-Mediated Glomerular Diseases
  • Th1 and Th17 Cells Induce Proliferative Glomerulonephritis
  • Toll-Like Receptor 2 Agonists Exacerbate Accelerated Nephrotoxic Nephritis
  • Anti-Neutrophil Cytoplasmic Antibodies and Effector CD4+ Cells Play Nonredundant Roles in Anti-Myeloperoxidase Crescentic Glomerulonephritis
  • Anti-Neutrophil Cytoplasm-Associated Glomerulonephritis
  • T Cells in Crescentic Glomerulonephritis
  • IL-12p40 and IL-18 in Crescentic Glomerulonephritis: IL-12p40 is the Key Th1-Defining Cytokine Chain, Whereas IL-18 Promotes Local Inflammation and Leukocyte Recruitment
  • An IL-12-Independent Role for CD40-CD154 in Mediating Effector Responses: Studies in Cell-Mediated Glomerulonephritis and Dermal Delayed-Type Hypersensitivity
  • Intrinsic Renal Cell Expression of CD40 Directs Th1 Effectors Inducing Experimental Crescentic Glomerulonephritis
  • T Cells and Minimal Change Disease
  • The Requirement for Granulocyte-Macrophage Colony-Stimulating Factor and Granulocyte Colony-Stimulating Factor in Leukocyte-Mediated Immune Glomerular Injury
  • Human Glomerulonephritis Accompanied by Active Cellular Infiltrates Shows Effector T Cells in Urine
  • Glomerulonephritis Induced by Recombinant Collagen IV{alpha}3 Chain Noncollagen Domain 1 Is Not Associated with Glomerular Basement Membrane Antibody: A Potential T Cell-Mediated Mechanism
  • Interleukin-12 from Intrinsic Cells Is an Effector of Renal Injury in Crescentic Glomerulonephritis
  • Expression of the Chemokine Monocyte Chemoattractant Protein-1 and Its Receptor Chemokine Receptor 2 in Human Crescentic Glomerulonephritis
  • Clinical Utility of Testing for Antineutrophil Cytoplasmic Antibodies
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