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Hormones, Growth Factors, Cell Biology and Structure
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A Conditionally Immortalized Human Podocyte Cell Line Demonstrating Nephrin and Podocin Expression

Moin A. Saleem, Michael J. O’Hare, Jochen Reiser, Richard J. Coward, Carol D. Inward, Timothy Farren, Chang Ying Xing, Lan Ni, Peter W. Mathieson and Peter Mundel
JASN March 2002, 13 (3) 630-638;
Moin A. Saleem
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Michael J. O’Hare
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Jochen Reiser
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Richard J. Coward
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Carol D. Inward
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Timothy Farren
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Chang Ying Xing
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Lan Ni
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Peter W. Mathieson
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Peter Mundel
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Article Information

vol. 13 no. 3 630-638
PubMed 
11856766

Published By 
American Society of Nephrology
Print ISSN 
1046-6673
Online ISSN 
1533-3450
History 
  • Received for publication July 14, 2001
  • Accepted for publication November 1, 2001
  • Published online March 1, 2002.

Copyright & Usage 
© 2002 American Society of Nephrology

Author Information

  1. Moin A. Saleem*,
  2. Michael J. O’Hare†,
  3. Jochen Reiser‡,
  4. Richard J. Coward*,
  5. Carol D. Inward*,
  6. Timothy Farren*,
  7. Chang Ying Xing*,
  8. Lan Ni*,
  9. Peter W. Mathieson* and
  10. Peter Mundel‡
  1. *Children’s Renal Unit and Academic Renal Unit, University of Bristol, Southmead Hospital, Bristol, UK; †LICR/UCL Breast Cancer Laboratory, London, UK; ‡Department of Medicine and Department of Anatomy and Structural Biology, Albert Einstein College of Medicine, Bronx, New York.
  1. Correspondence to: Dr. Moin A. Saleem, Children’s Renal Unit and Academic Renal Unit, University of Bristol, Southmead Hospital, Bristol, BS10 5NB UK. Phone: +44-117-959-6048; Fax: +44-117-959-5438; E-mail: M.Saleem{at}bristol.ac.uk
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Journal of the American Society of Nephrology: 13 (3)
Journal of the American Society of Nephrology
Vol. 13, Issue 3
1 Mar 2002
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A Conditionally Immortalized Human Podocyte Cell Line Demonstrating Nephrin and Podocin Expression
Moin A. Saleem, Michael J. O’Hare, Jochen Reiser, Richard J. Coward, Carol D. Inward, Timothy Farren, Chang Ying Xing, Lan Ni, Peter W. Mathieson, Peter Mundel
JASN Mar 2002, 13 (3) 630-638;

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A Conditionally Immortalized Human Podocyte Cell Line Demonstrating Nephrin and Podocin Expression
Moin A. Saleem, Michael J. O’Hare, Jochen Reiser, Richard J. Coward, Carol D. Inward, Timothy Farren, Chang Ying Xing, Lan Ni, Peter W. Mathieson, Peter Mundel
JASN Mar 2002, 13 (3) 630-638;
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  • Sphingosine-1-Phosphate Metabolism and Signaling in Kidney Diseases
  • LIM-Nebulette Reinforces Podocyte Structural Integrity by Linking Actin and Vimentin Filaments
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  • GPRC5b Modulates Inflammatory Response in Glomerular Diseases via NF-{kappa}B Pathway
  • Overexpression of Circulating Soluble Nogo-B Improves Diabetic Kidney Disease by Protecting the Vasculature
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  • Thrombospondin Type 1 Domain-Containing 7A Localizes to the Slit Diaphragm and Stabilizes Membrane Dynamics of Fully Differentiated Podocytes
  • Integrated Functional Genomic Analysis Enables Annotation of Kidney Genome-Wide Association Study Loci
  • Treatment with 2,4-Dihydroxybenzoic Acid Prevents FSGS Progression and Renal Fibrosis in Podocyte-Specific Coq6 Knockout Mice
  • Recruitment of APOL1 kidney disease risk variants to lipid droplets attenuates cell toxicity
  • Manipulation of Nephron-Patterning Signals Enables Selective Induction of Podocytes from Human Pluripotent Stem Cells
  • The long noncoding RNA LOC105374325 causes podocyte injury in individuals with focal segmental glomerulosclerosis
  • Histone H3 Serine 10 Phosphorylation Facilitates Endothelial Activation in Diabetic Kidney Disease
  • Increased autophagy is cytoprotective against podocyte injury induced by antibody and interferon-{alpha} in lupus nephritis
  • Induced Pluripotent Stem Cell-Derived Podocyte-Like Cells as Models for Assessing Mechanisms Underlying Heritable Disease Phenotype: Initial Studies Using Two Alport Syndrome Patient Lines Indicate Impaired Potassium Channel Activity
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