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Cell Biology
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Angiotensin II Type 1–Receptor Mediated Changes in Heparan Sulfate Proteoglycans in Human SV40 Transformed Podocytes

Paul-Thomas Brinkkoetter, Simone Holtgrefe, Fokko J. van der Woude and Benito A. Yard
JASN January 2004, 15 (1) 33-40; DOI: https://doi.org/10.1097/01.ASN.0000102476.50041.44
Paul-Thomas Brinkkoetter
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Simone Holtgrefe
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Fokko J. van der Woude
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Benito A. Yard
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Article Information

vol. 15 no. 1 33-40
DOI 
https://doi.org/10.1097/01.ASN.0000102476.50041.44
PubMed 
14694155

Published By 
American Society of Nephrology
Print ISSN 
1046-6673
Online ISSN 
1533-3450
History 
  • Received for publication October 23, 2002
  • Accepted for publication September 7, 2003
  • Published online December 23, 2003.

Copyright & Usage 
© 2004 American Society of Nephrology

Author Information

  1. Paul-Thomas Brinkkoetter,
  2. Simone Holtgrefe,
  3. Fokko J. van der Woude and
  4. Benito A. Yard
  1. V. Medizinische Universitätsklinik, Klinikum Mannheim, University of Heidelberg, Heidelberg, Germany
  1. Correspondence to Dr. Benito A. Yard, V. Medizinische Universitätsklinik, Klinikum Mannheim, University of Heidelberg, Theodor-Kutzer-Ufer 1-3, 68135 Mannheim, Germany. Phone: 0049-621-3832340; Fax: 0049-621-3833804;
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Journal of the American Society of Nephrology: 15 (1)
Journal of the American Society of Nephrology
Vol. 15, Issue 1
1 Jan 2004
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Angiotensin II Type 1–Receptor Mediated Changes in Heparan Sulfate Proteoglycans in Human SV40 Transformed Podocytes
Paul-Thomas Brinkkoetter, Simone Holtgrefe, Fokko J. van der Woude, Benito A. Yard
JASN Jan 2004, 15 (1) 33-40; DOI: 10.1097/01.ASN.0000102476.50041.44

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Angiotensin II Type 1–Receptor Mediated Changes in Heparan Sulfate Proteoglycans in Human SV40 Transformed Podocytes
Paul-Thomas Brinkkoetter, Simone Holtgrefe, Fokko J. van der Woude, Benito A. Yard
JASN Jan 2004, 15 (1) 33-40; DOI: 10.1097/01.ASN.0000102476.50041.44
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More in this TOC Section

  • Lack of Connexin 40 Causes Displacement of Renin-Producing Cells from Afferent Arterioles to the Extraglomerular Mesangium
  • Antineutrophil Cytoplasm Antibody–Stimulated Neutrophil Adhesion Depends on Diacylglycerol Kinase–Catalyzed Phosphatidic Acid Formation
  • Human T and Natural Killer Cells Possess a Functional Renin-Angiotensin System: Further Mechanisms of Angiotensin II–Induced Inflammation
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  • Mechanisms of Metabolic Acidosis-Induced Kidney Injury in Chronic Kidney Disease
  • Angiotensin Receptor Blocker Protection Against Podocyte-Induced Sclerosis Is Podocyte Angiotensin II Type 1 Receptor-Independent
  • Angiotensin II Type 1 Receptor Blockade Inhibits the Development and Progression of HIV-Associated Nephropathy in a Mouse Model
  • Proteinuria-Lowering Effect of Heparin Therapy in Diabetic Nephropathy without Affecting the Renin-Angiotensin-Aldosterone System
  • Renin-Angiotensin-Aldosterone System and Progression of Renal Disease
  • Induction of Glomerular Heparanase Expression in Rats with Adriamycin Nephropathy Is Regulated by Reactive Oxygen Species and the Renin-Angiotensin System
  • From the Periphery of the Glomerular Capillary Wall Toward the Center of Disease: Podocyte Injury Comes of Age in Diabetic Nephropathy
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