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BASIC RESEARCH
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Preconditioning with Endoplasmic Reticulum Stress Ameliorates Mesangioproliferative Glomerulonephritis

Reiko Inagi, Takanori Kumagai, Hiroshi Nishi, Takahisa Kawakami, Toshio Miyata, Toshiro Fujita and Masaomi Nangaku
JASN May 2008, 19 (5) 915-922; DOI: https://doi.org/10.1681/ASN.2007070745
Reiko Inagi
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Takanori Kumagai
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Hiroshi Nishi
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Takahisa Kawakami
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Toshio Miyata
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Toshiro Fujita
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Masaomi Nangaku
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Article Information

vol. 19 no. 5 915-922
DOI 
https://doi.org/10.1681/ASN.2007070745
PubMed 
18256359

Published By 
American Society of Nephrology
Print ISSN 
1046-6673
Online ISSN 
1533-3450
History 
  • Received for publication July 9, 2007
  • Accepted for publication November 29, 2007
  • Published online April 28, 2008.

Article Versions

  • Earlier version (February 6, 2008 - 12:48).
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© 2008 American Society of Nephrology

Author Information

  1. Reiko Inagi*,
  2. Takanori Kumagai*,
  3. Hiroshi Nishi*,
  4. Takahisa Kawakami*,
  5. Toshio Miyata†,
  6. Toshiro Fujita* and
  7. Masaomi Nangaku*
  1. *Division of Nephrology and Endocrinology, University of Tokyo School of Medicine, Tokyo, and †Department of Internal Medicine, Tokai University School of Medicine, Kanagawa, Japan
  1. Correspondence:
    Dr. Reiko Inagi, Division of Nephrology and Endocrinology, University of Tokyo School of Medicine, 7-3-1, Hongo Bunkyo-ku, Tokyo 113-8655, Japan. Phone: +81-3-3815-5411; Fax: +81-3-5800-8806; E-mail: inagi-npr{at}umin.ac.jp
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Journal of the American Society of Nephrology: 19 (5)
Journal of the American Society of Nephrology
Vol. 19, Issue 5
May 2008
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Preconditioning with Endoplasmic Reticulum Stress Ameliorates Mesangioproliferative Glomerulonephritis
Reiko Inagi, Takanori Kumagai, Hiroshi Nishi, Takahisa Kawakami, Toshio Miyata, Toshiro Fujita, Masaomi Nangaku
JASN May 2008, 19 (5) 915-922; DOI: 10.1681/ASN.2007070745

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Preconditioning with Endoplasmic Reticulum Stress Ameliorates Mesangioproliferative Glomerulonephritis
Reiko Inagi, Takanori Kumagai, Hiroshi Nishi, Takahisa Kawakami, Toshio Miyata, Toshiro Fujita, Masaomi Nangaku
JASN May 2008, 19 (5) 915-922; DOI: 10.1681/ASN.2007070745
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  • Restoration of Proteostasis in the Endoplasmic Reticulum Reverses an Inflammation-Like Response to Cytoplasmic DNA in Caenorhabditis elegans
  • Activation of the endoplasmic reticulum unfolded protein response reverses an inflammation-like response to cytoplasmic DNA
  • Quantifying multi-layered expression regulation in response to stress of the endoplasmic reticulum
  • Cold Shock Proteins Mediate GN with Mesangioproliferation
  • Phenotypic assays identify azoramide as a small-molecule modulator of the unfolded protein response with antidiabetic activity
  • Bioinformatics Analysis of Proteomic Profiles During the Process of Anti-Thy1 Nephritis
  • Interrelationship Between Cardiac Hypertrophy, Heart Failure, and Chronic Kidney Disease: Endoplasmic Reticulum Stress As a Mediator of Pathogenesis
  • Preconditioning with Endoplasmic Reticulum Stress Mitigates Retinal Endothelial Inflammation via Activation of X-box Binding Protein 1
  • ER Stress Depresses NF-{kappa}B Activation in Mesangial Cells through Preferential Induction of C/EBP{beta}
  • Endoplasmic Reticulum Stress in Glomerulonephritis: The Bad Guy Turns Good?
  • A Subcytotoxic Dose of Subtilase Cytotoxin Prevents Lipopolysaccharide-Induced Inflammatory Responses, Depending on its Capacity to Induce the Unfolded Protein Response
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