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Experimental insights into the tubulointerstitial disease accompanying primary glomerular lesions.

A A Eddy
JASN December 1994, 5 (6) 1273-1287;
A A Eddy
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Article Information

vol. 5 no. 6 1273-1287
PubMed 
7893992

Published By 
American Society of Nephrology
Print ISSN 
1046-6673
Online ISSN 
1533-3450
History 
  • Published online December 1, 1994.

Copyright & Usage 
Copyright © 1994 by American Society of Nephrology

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  1. A A Eddy

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Journal of the American Society of Nephrology
Vol. 5, Issue 6
1 Dec 1994
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Experimental insights into the tubulointerstitial disease accompanying primary glomerular lesions.
A A Eddy
JASN Dec 1994, 5 (6) 1273-1287;

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Experimental insights into the tubulointerstitial disease accompanying primary glomerular lesions.
A A Eddy
JASN Dec 1994, 5 (6) 1273-1287;
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  • Role of Extracellular Matrix Renal Tubulo-interstitial Nephritis Antigen (TINag) in Cell Survival Utilizing Integrin {alpha}v{beta}3/Focal Adhesion Kinase (FAK)/Phosphatidylinositol 3-Kinase (PI3K)/Protein Kinase B-Serine/Threonine Kinase (AKT) Signaling Pathway
  • Hyaluronan Facilitates Transforming Growth Factor-{beta}1-dependent Proliferation via CD44 and Epidermal Growth Factor Receptor Interaction
  • Hypothesis: Dent Disease Is an Underrecognized Cause of Focal Glomerulosclerosis
  • Resolvin D Series and Protectin D1 Mitigate Acute Kidney Injury
  • Glomerulotubular Junction Abnormalities Are Associated with Proteinuria in Type 1 Diabetes
  • DNA Vaccination with CCL2 DNA Modified by the Addition of an Adjuvant Epitope Protects against "Nonimmune" Toxic Renal Injury
  • Chronic Hypoxia and Tubulointerstitial Injury: A Final Common Pathway to End-Stage Renal Failure
  • Hepatocyte Growth Factor Modulates Matrix Metalloproteinases and Plasminogen Activator/Plasmin Proteolytic Pathways in Progressive Renal Interstitial Fibrosis
  • Reversal of Glomerulosclerosis after High-Dose Enalapril Treatment in Subtotally Nephrectomized Rats
  • Chronic Progression of Tubulointerstitial Damage in Proteinuric Renal Disease Is Mediated by Complement Activation: A Therapeutic Role for Complement Inhibitors?
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  • Atubular Glomeruli and Glomerulotubular Junction Abnormalities in Diabetic Nephropathy
  • C6 Mediates Chronic Progression of Tubulointerstitial Damage in Rats with Remnant Kidneys
  • Lacidipine Inhibits Adhesion Molecule and Oxidase Expression Independent of Blood Pressure Reduction in Angiotensin-Induced Vascular Injury
  • Bone Morphogenetic Protein-7 Improves Renal Fibrosis and Accelerates the Return of Renal Function
  • Glomerular Injury and Tubular Loss in Adriamycin Nephrosis
  • Specific Antagonism of PDGF Prevents Renal Scarring in Experimental Glomerulonephritis
  • Expression of Apoptosis-Related Genes and Proteins in Experimental Chronic Renal Scarring
  • Recommendations for the Outpatient Surveillance of Renal Transplant Recipients
  • Induction of CD14 in Tubular Epithelial Cells During Kidney Disease
  • Contribution of Angiotensin II to Late Renal Injury after Acute Ischemia
  • Course of Renal Injury in the Mpv17-Deficient Transgenic Mouse
  • Complement Activation Products in the Urine from Proteinuric Patients
  • Effect of Angiotensin-Converting Enzyme Inhibition on Glomerular Basement Membrane Permeability and Distribution of Zonula Occludens-1 in MWF Rats
  • Neutral Lipid from Proteinuric Rat Urine Is a Novel Inhibitor of the Red Blood Cell Calcium Pump
  • Expansion of Cortical Interstitium Is Limited by Converting Enzyme Inhibition in Type 2 Diabetic Patients with Glomerulosclerosis
  • Induction of Monocyte Chemoattractant Protein-1 by Albumin Is Mediated by Nuclear Factor {kappa}B in Proximal Tubule Cells
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